Cardiodynamic and electrocardiographic changes in normal pregnancy

Cardiodynamic and electrocardiographic changes in normal pregnancy

353 ABSTRACTS fourth to the seventh month, and furthermore eighth and ninth months, without having the isonumeric red corpuscles, the author suggests...

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353

ABSTRACTS fourth to the seventh month, and furthermore eighth and ninth months, without having the isonumeric red corpuscles, the author suggests a.ctivity.

show progressive increase said volume reach that of that pregnancy stimulates MARIO

Landt and Benjamin: Cardiodynamic and Electrocardiographic Pregnancy, Am. Heart J. 12: 592, 1936.

A.

during the the normal microcytic CASTALLO.

Changes in Normal

Observations were made on 19 normal pregnant women throughout the course of their pregnancy and puerperium. The study included correlation of clinical, cardioFinal examination was made approxidynamic, and electrocardiographic changes. mately two months after delivery. The study indicates that pregnancy definitely places a burden on the cardiocirculatory system. In the normal woman this burden can be compensated by calling on the reserve capacity of this system. The method of compensation is both mechanical and physiologic. The electrocardiographic changes during pregnancy may be interpreted definitely on the basis of mechanical shifting of the heart, usually expressed in a left axis deviation in the electrocardiogram. The normal woman who does not develop any untoward signs or symptoms during the course of pregnancy shows a normal clinical, cardiodynamic and eleotrocardiographic picture six to eight weeks after delivery. J. P. GREENHILL.

Burwell, C. Sidney: The Placenta as a Modified Arteriovenous Fistula, Considered in Relation to the Circulatory Adjustments to Pregnancy, Am. J. M. SC. 195: 7, 1938. The demonstrated phenomena of the circulation in pregnant women plus the evidence offered by the structure of the placenta lead to the conclusion, that the changes in the circulation during pregnancy are in the main to be as&bed to two mechanisms: (1) Obstruction to venous return by the enlarged uterus ; (2) an arteriovenous leak through the placenta. J. THORNWELL WITHERSPOON.

Piccone, L.: The Larynx 131, 1935.

in Pregnancy,

Folia

Gynaec.-Demograph.

(Genova)

31:

The author refers to previous studies of this question. His histologic studies on dogs and rabbits demonstrated common changes in all the organs of the body and certain slighter modifications limited to special areas, particularly glottis and epiglottis. In the larynx, can be observed new formation of blood vessels, a hyperemia of the vessels themselves, together with a more or less marked lymphatic infiltration. The connective tissue is found to be edematous with lymphatic spaces greatly dilated. These changes were not found in the nonpregnant control animals nor in animals subjected to hormonal injections. These processes, the author feels may explain the rapid progress of laryngeal tuberculous lesions during pregnancy. MARIO A. CASTALLO.

Abbot,& A. Clifford, and Prendergast, James: The Histology in Pregnancy, Canad. M. A. J. 34: 609, 1936.

of the Thyroid

Gland

The three principal beliefs regarding the thyroid gland in pregnancy are: (1) that the thyroid gland becomes hyperplastic during pregnancy, (2) that the thyroid gland becomes hypoplastic or colloid in nature during pregnancy, (3) that there is no change in the histology of the thyroid gland. In general, American observers favor the hyperplastic theory, while continental workers believe tha,t there is no As yet, there is no definite change or that the gland becomes colloid in nature. proof of pathologic change.